Coloring Picture For Guidance Method For Children: Study Introduction In Sick Child Recovery Program

Coloring Picture For Guidance Method For Children: Study Introduction In Sick Child Recovery Program

Abstract
Coloring Book Picture is a creative play therapy to reduce stress and anxiety and improve communication in children.

Purpose
to evaluate changes in behavior (eating, receiving medical actions and communications) on the Recovery Program Sick Children Hospital Dr. Soetomo Surabaya after receiving therapeutic interventions creative games using the media Picture Book.

Patients and methods
Experimental studies (pre and post), sample: 10 patients who were treated at the children's ward Dr hospitals. Soetomo Surabaya, during the months of January to February 1999. Media Books Image of situations and conditions for treatment in hospital, including diagnostic and therapeutic procedures. Statistical analysis using Chi Square test and Fischer Exact Test.

Results
8 patients (n = 10) obtained a positive change in behavior after the intervention (X2 = 3.6 DF.1 p

Conclusion Coloring Book Illustrated as creative play therapy is a method of health education to change the behavior of children during hospitalization

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spinal cord tumor

I. DEFINITIONS
Spinal cord tumor is a tumor that develops in the spine or its contents and usually cause symptoms because of the involvement of the spinal cord or nerve roots. (Price sylvia anderson, 1995)

II.KLASIFIKASIONS
a.Tumor Intradural
In contrast to intradural tumors ekstradural tumors generally benign.
Tumor Ekstramedular
Durameter and located between the spinal cord, most of the tumors in this region are benign neurofibroma or meningioma
Tumor Intramedular
Derived from the spinal cord itself.

b.Tumor Ekstradural
Tumor ekstradural primarily metastase from primary lesions in the breast, prostate, thyroid, lung, kidney, and stomach
Tumor ekstradural generally come from the vertebral column or from the room ekstradural. Neoplasm ekstradural indoor ekstradural carcinoma and lymphoma usually metastase.

III.MANIFESTASI OF CLINIC
Tumor ekstradural
Pain is described as a constant and limited to the tumor area followed by a pain that extends dermatome pattern
Greatest pain at night and become more powerful by the movement of the spine and the rest lie
Radikuler pain exacerbated by coughing and straining
The pain may last for several weeks or months prior to the involvement of the spinal cord.
Spinal cord function will vanish altogether
Spastic weakness and loss of vibration sensation
Parestesi and sensory deficits will grow rapidly into the irreversible paraplegia
Irritable bowel and urination

Intradural tumor
Clinical journey more slowly and lasted for months.
Reduced perception of pain and temperature below the level of the contralateral lesion
Patient complains of pain, at first on the back and then along the spinal roots
The pain intensified by movement, coughing, sneezing, or straining, and most severe at night (pain at night is caused by traction on the roots of the illness, which is when the spine is elongated after the disappearance of the effect of gravitational contraction.
Paraesthesia and continuing proprioceptive sensory deficit

IV.ETIOLOGI
Risk factors tumors can occur in any race group, the incidence increases with age, risk factors will increase in people exposed to certain chemicals (Okrionitil, inks, solvents, lubricating oils), but it is not. The influence of genetic tibulnya participate in tumors, tuberculosis and multiple sclerosis disease neurofibomatosis disease.

V.patofisiologi
Pathophysiological conditions due to spinal cord tumors are caused by damage and infiltration, shift and decompress the spinal cord and interruption of blood supply or cerebrospinal fluid. Degree of symptoms depending on the level of decompression, and speed the development, adaptation can occur with slow-growing tumors, 85% benign spinal cord tumors.
Especially good tumor arising neoplasms ekstramedula or intra-medullary. Secondary tumors or tumors can also interfere metastase the spinal cord and the lining and the vertebra
Ekstramedular tumor from tumor edge intramedural initially loaded the root causes of subjective pain. With the growth of tumors can arise motor and sensory deficits associated with the level of the spinal cord akardan attacked. Because there was a tumor growing emphasis on the spinal cord. In line with that of patients lose all motor function below the lesion and sensory / tumor

Spinal cord tumors, which starts from the spinal cord, often causing symptoms such as the central spinal cord, including segmental loss of pain and temperature functions. Moreover, the function of cells, anterior horn is often missing, especially on the hands. The whole point is near the central gray objects into dysfunction. Rasanyeri and sensory loss of temperature and motor weakness progress little by little, gain weight and decrease. Cauda motor and sensory functions of the latter will be lost, including the loss function fecal and urine elimination. (Long C, Barbara, 1996)
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Migraine

Autonomik dysfunction of blood vessel growth resulted head skin headache known as migraine. Actual mechanism of migraine is not all clear. But many factors - factors that prodram early menungkapkan of migraine must be related to the Vaso constriction of intra-cranial artery.
Symptoms are typical at this early stage is the emergence of skotoma and pale face.

Prodrom was followed by the emergence of sesisi headache and facial flushing. Soon after arising vomiting - vomiting, edema of nasal mucous membranes, fingers - fingers and toes.
Symptoms are regarded as a manifestation of stage vasodolatasi extra cranial arteries.
What causes dysfunction of blood vessels is still unknown, but probably once a congenital disorder, due to familial and hereditary factors certainly have the migraine. (Prof. Dr. Mahar Mardjono, Clinical Neurology)

Among the many types of headache, migraine is a type of the most researched and discussed, in addition to the causes which are still mysteries, then the incident is enough to encourage the experts to examine it.

Aretaeusi (80 AD) is one of the researchers in his day headache and it was he who first described the symptoms of headache have distinctive profile. He introduced this type with the name "HETEROCRANIA" which means headache.
By Galen 50 years later changed to "HEMICRANIA" and then the experts from the French to change again into their language as "MEGRIM" for details said - he said the "migraine" (Dr. Sidiarto. K)


A. anxiety / worry
Definition
Anxiety or anxiety is a vague feeling of fear - a vague source, are often not specific or not known by that individual.
Anxiety is the feeling / emotional response to the assessment, feeling uncertain and powerless (Stuart and Sundeen, 1988). Experienced emotional state objectively and communicated in interpersonal relationships. Anxiety is an emotional response to the assessment of daily life - today. Describes the state of anxiety worry, anxiety, fear, not peaceful with various physical kekuhan.

Predisposing factors

1. Analytic psycho theory
According to Freud, personality structure consists of 3 elements is "ID, EGO, & SUPER EGO". Symbolizes the ego instincts and impulses urge primiti, Super Ego represents the conscience of a person and controlled by the norm - a cultural norm. While diagambarkan ego as a mediator between the demands of the ID & Super Ego.

2. INTERPERSONAL theory
Anxiety occurs from the fear of interpersonal rejection. It is also associated with trauma to the growth, such as loss, separation of individuals who have low self-esteem is usually very easy to experience severe anxiety.

3.BEHAVIOR theory
Frusatasi anxiety is the result of everything that interfere with a person's ability to achieve the desired objectives.
This theory believes that humans are exposed early in life in an excessive fear will indicate the possibility of severe anxiety in the lives of his adult life.

Anxiety Light
Connect with mild anxiety tension events of everyday life. At this level of perception of land to widen and individuals will be aware - and wary hearts.

Physiological Response
Occasional shortness of breath
Tone and blood pressure rise
Mild symptoms of the stomach
Wrinkled face and trembling lips

Cognitive Response
Able to accept the complex stimuli
Concentrate on problem
Solve problems effectively

Response Behavior and Emotions
Unable to sit still
Fine tremor of the hands
Sometimes sound - sometimes rising

Anxiety was
At this level of land decreased perception of the environment, the individual is more important to focus on the moment and the exclusion of other things.


Physiological response
Often short of breath
Pulse and blood pressure rise
Dry mouth
Anorexia
Diarrhea / constipation
Restless
Cognitive response
Perceptual field narrows
External stimuli can not be accepted
Focus on what is a concern
Behavioral and emotional responses
Movement jerk - jerk / squeezed
Talk more and more quickly
Difficulty sleeping
Insecurity

Serious anxiety
In severe anxiety land becomes very narrow perception and then incapable of thinking.
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BRAIN TUMOR

Definition:
Brain tumor is intra-cranial lesions that occupy space in the skull

Classification of Brain Tumors:
1.Tumor from lapisam brain (meningioma dural)
2.Tumor growing in / on the cranial nerves
3.Tumor originating in the brain tissue
4.Lesi metastatic from any body part

Pathophysiology:
Brain tumor causing neurological disorders. The symptoms occur sequentially. This emphasizes the importance of anamnesis in the examination of the client. Symptoms should be discussed within a perspective of time.
Neurologic symptoms of brain tumors are usually considered to be caused by 2 factors focal disruption, caused by the tumor and intracranial pressure. Focal disruption occurs when the emphasis on the brain tissue and infiltration / invasion directly on the brain parenchymal tissue damage neurons. Of course the greatest dysfunction occurs in tumors that grow most rapidly.
Changes in blood supply due to pressure generated tumors that grow brain tissue necrosis. Arterial blood supply disruptions generally manifests as an acute loss of function and may be confused with primary cerebrovaskuler disorders. Seizures as a manifestation of neuro sensitivity changes associated with invasion and compression of the blood supply to changes in brain tissue. Some tumors also form cysts around the brain parenchymal pressure so that aggravate focal neurological disorders.

Increased intra-cranial pressure can be caused by several factors: the increase of mass in the skull, the formation of edema around the tumor and cerebrospinal circulation changes. Growth of the tumor mass will increase, because the tumor will take space from the relatively rigid skull. Malignant tumor jaruingan cause edema in the brain. Seluruhnyanya not yet understood mechanism, but due to the difference in osmotic allegedly causing bleeding. Venous obstruction and edema caused by damage to blood brain barrier, it caused an increase in intracranial volume. Observation of cerebrospinal fluid circulation from the ventricles laseral into hidrocepalus sub arakhnoid cause.

Increased intracranial pressure would endanger life, when there is rapid due to one of the causes that have been discussed previously. Compensation mechanisms need time to become effective berhari-hari/berbulan-bulan and oelh because ity was not useful when rapid intracranial pressure arise. These compensatory mechanisms include working lower intra-cranial blood volume, cerebrospinal fluid volume, intracellular fluid content and reduce parenchymal cells. The increase in pressure resulted in untreated ulcer or serebulum herniation. Herniation occurs when the medial lobe girus shifted to the inferior temporals through tentorial notch by the masses in the brain hemispheres. Herniation pressing ensefalon menyebabkab to loss of consciousness and menenkan third nerve. In serebulum herniation, tonsils before the shift down through the foramen magnum by a posterior mass. Compression of the medulla oblongata and stopped breathing happen quickly. Intracranial bradicardi fast is a progressive, systemic hypertension (widening pulse pressure and respiratory problems).

Signs and Symptoms
According to tumor location:
1.Lobus frontalis
Mental disorder / mild personality disorders: depression, confusion, strange behavior, it is difficult to give argumenatasi / assess true or not, hemiparesis, ataxia, and disturbances to speak.
Posterior 2.Kortek presentalis
Weakness / paralysis of facial muscles, tongue and fingers
3.Lobus parasentralis
Weakness in the lower limb
4.Lobus Oksipitalis
Seizures, vision disturbances
5.Lobus temporalis
Tinnitus, auditory hallucinations, sensory Aphasia, facial muscle paralysis
6.Lobus parietal
Missing sensory function, kortikalis, localization sensory disturbances, visual impairment
7.Cerebulum
Papil edema, headache, motor disturbances, hipotonia, hiperekstremitas esndi

Common signs and symptoms:
1.Nyeri heavy head in the morning, play increased when coughing, bending
2.Kejang
3.Tanda-mark intra-cranial pressure: blurred vision, nausea, vomiting, decreased auditory function, changes in vital signs, Aphasia.
4.Changes of personality
Memory
5.Perasaa natural


Classical triad;
Headache
Papil edema
Vomiting

Diagnostic Examination;
1.Rontgent anterior-posterior skull
2.EEG
3.CT Scan
4.MRI
5.Angioserebral
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Meningitis Desease

A. Definitions
Meningitis is an inflammation of the meningen (membrane that surrounds the brain and spinal cord) and is caused by viruses, bacteria or fungi organs (Smeltzer, 2001).
Meningitis is an acute infection of the meninges, usually caused by one of the microorganisms pneumokok, Meningokok, Stafilokok, Streptokok, Hemophilus influenza and materials aseptic (viral) (Long, 1996).

Meningitis is an inflammation of the membrane meningen, cerebrospinal fluid and spinal column that causes the infection in the central nervous system (Suriadi & Rita, 2001).

B. Etiology
1.Bakteri; Mycobacterium tuberculosa, Diplococcus pneumoniae (pneumokok), Neisseria meningitis (meningokok), Streptococus haemolyticuss, Staphylococcus aureus, Haemophilus influenzae, Escherichia coli, Klebsiella pneumoniae, Peudomonas aeruginosa
Other
2.Penyebab Lues, Virus, Toxoplasma gondhii and Ricketsia
3.Predisposing factors: gender men, more often than women
4.maternal factor : fetal membrane rupture, maternal infections during the last week of pregnancy
5.immunology factors : immune mechanism deficiency, immunoglobulin deficiency.
6.Kelainan central nervous system, surgery or injury associated with the neural system


C. Classification
Meningitis is divided into 2 groups based on changes in brain fluid, namely:
1.Meningitis serous
Meningitis is araknoid and pia mater of the brain accompanied by a clear fluid. The cause is terseringnya Mycobacterium tuberculosa. Other causes Lues, Virus, Toxoplasma gondhii and Ricketsia.
2.Meningitis purulenta
Purulent inflammation is arakhnoid and the pia mater covering the brain and spinal cord. The reasons include: Diplococcus pneumoniae (pneumokok), Neisseria meningitis (meningokok), Streptococus haemolyticuss, Staphylococcus aureus, Haemophilus influenzae, Escherichia coli, Klebsiella pneumoniae, Peudomonas aeruginosa.

C. Pathophysiology
Begins as a bacterial meningitis infection from oroaring and followed by septicemia, which meningen spread to the brain and upper spinal cord.
Predisposing factors include infection of the upper airway, otitis media, mastoiditis, sickle cell anemia and other hemoglobinopatis, a new neurosurgical procedures, head trauma and immunological effects. Venous channels through the posterior nasopharynx, middle ear and mastoid channel close to the brain and the veins channel meningen; all of this link which underpins the development of bacteria.
Organisms into the bloodstream and cause inflammation in the meningen and under the cortex, which can cause a thrombus and decreased cerebral blood flow. Cerebral tissue due to impaired metabolism meningen exudate, vasculitis and hipoperfusi. Purulent exudate may spread to the base of the brain and spinal cord. Inflammation also spread to the membrane walls of cerebral ventricles. Bacterial meningitis associated with intracranial physiological changes, which consisted of increased permeability of the blood, brain defense areas (barrier oak), cerebral edema and improvement of ICT.
In acute infection patients died of bacterial toxins before the meningitis. Most infections of patients with adrenal damage, circulatory collapse and associated with widespread hemoragi (at sindromWaterhouse-Friderichssen) as a result of endothelial damage and necrosis of blood vessels caused by the meningococcus.

D. Clinical Manifestations
Symptoms of meningitis resulting from infection and increased ICT:
1.head pain and fever (often initial symptoms)
2.Changes at the level of consciousness can occur letargik, unresponsive, and coma.
3.Iritasi meningen caused some signs as follows:
a) nukal rigidity (stiff neck). Attempts to head flexion in difficulty due to spasm of the neck muscles.
b) kernik positive sign: when the patient lay with the state of flexion of the thigh towards the abdomen, legs can not be in perfect ekstensikan.
c) Signs brudzinki: if the patient's neck in flexion produced reflect the knees and hips. When done on passive flexion of the lower limb on one side of the same movement seen bike ektremita opposite side.
4. get photo phobias, or excessive sensitivity to light.
5.Kejang due to focal cortical areas are sensitive and improvement of ICT by purulent exudate and cerebral edema with signs of the characteristic changes of vital signs (pulse pressure and widening bradikardi), irregular breathing, headaches, vomiting and decreased level of consciousness.
6.conspicuous rash is characteristic of meningococcal meningitis.
7.fulminating infections with septikimia signs: high fever suddenly appeared, lesions that spread purpura, shock and signs of disseminated coagulopathy intravaskuler

E. Diagnostic Examination
CSS 1.Analisis of lumbar function:
a) Bacterial meningitis: increased pressure, the liquid cloudy / foggy, white blood cell count and protein increased glucose increased, a positive culture for several types of bacteria.
b) viral meningitis: the pressure varies, CSS is usually clear liquid, white blood cells, glucose and protein is usually normal, the culture is usually negative, viral culture is usually with a special procedure.
2.Glukosa serum: increased (meningitis)
3.LDH serum: increased (bacterial meningitis)
White blood 4.Sel: slightly increased with the increase in neutrophils (bacterial infection)
Blood 5.Elektrolit: Abnormal.
6.ESR/LED: increases in meningitis
Blood 7.Kultur / nose / throat / urine: may indicate the central region indicates the type of infection or infectious
8.MRI / CT scan: may be helpful in localize lesions, see the size / location of the ventricles; regional cerebral hematoma, hemorrhagic, or tumors
9.Ronsen chest / head / sinus; be no indication of the source of intra-cranial infection.

F. Complications
1.Hidrosefalus obstructive
2.MeningococcL septicemia (mengingocemia)
3.Sindrome water-friderichen (septic shock, DIC, bilateral adrenal hemorrhage)
4.SIADH (Syndrome Inappropriate Antidiuretic hormone)
5.Efusi subdural
6.Kejang
7.Edema and cerebral herniation
8.Cerebral palsy
Mental 9.Gangguan
10.Gangguan learning
11.Attention deficit disorder
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NON HEMORAGIK Stroke

A. Definitions
Circulatory disturbances diotak (GPDO) or known as the CVA (Cerebro Vaskuar Accident) is the function of nerve interference caused by the interruption of blood flow in the brain that can occur in a sudden (in seconds) or rapidly (within several hours) with symptoms or signs in accordance with the subject area. (Harsono, 1996, p. 67)

Stroke is cerebrovaskuler injury or loss of brain function caused by berhentinya supply blood to the brain is often serebrovaskuler during the culmination of several years. (Suzanne C. Smeltzer, 2002, p. 2131)

This disease is the third ranking cause of death in the United State. Due to stroke in each age level, but most often at the age between 75 - 85 years. (C Long., Barbara, 1996, p. 176).

B. Etiology
The cause-the cause of:
1.Trombosis (clot in the fluid in the brain blood vessel)
Cerebral 2.Embolisme (blood clot or other materials)
3.Iskemia (decrease blood flow to areas of the brain)
(Suzanne C. Smeltzer, 2002, p. 2131)

C. The risk of stroke
1.Hipertension
2.kardiovaskuler desease : arteria koronaria, heart failure kongestif, atrium fibrillation, heart disease kongestif)
3.Kolesterol high
4.Obesity
5.increasing hematokrit (risk infark serebral)
6.Diabetes Melitus (relating to aterogenesis terakselerasi)
7.oral contrasepsions (especially with hypertension, merkok, and high estrogen content)
8.drugs (cocaine)
9.alcohol
(Suzanne C. Smeltzer, 2002, p. 2131)

D. Clinical manifestation
Symptoms - symptoms appear CVA due to a particular region is not functioning because of the erorr of blood flow to the place. Symptoms that appear vary depending of the erorr.symtoms part of the brain-the symptoms are, among others:

a while
Sebebtar arise only for a few minutes to several hours and lost its own with or without treatment. This is called a transient ischemic attack (TIA). Attacks can appear again in the same form, make, or even live.

b. a whilel, but more than 24 hours
Symptoms occur more than 24 hours and this dissebut reversible ischemic neurologic deficit (RIND)

c. the longer the weight of the symptoms (progressive)
This desebabkan interruption of blood flow the longer the weight dissebut progressing stroke or stroke inevolution

d. permanent

(Harsono, 1996, p. 67)
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LOW BACK PAIN

A. Definition
Sensori is painful and emotional experience that is not enjoyable as a result of damage to the network and the actual potential. Definition of nursing is painful, painful for any of the body of the individual / mengalaminya someone who, whenever there is a person you say it (2). Regulations in primary care for patients with pain is that all pain is real, although it is not known. Therefore, the presence of pain is based only on patient reports.
Low Back Pain (LBP) or the bottom of the back pain is a painful sensation that is felt in the discus intervertebralis generally lumbal down, L4-L5 and L5-S1 (2.4).

B. Etiology
Most back pain is caused by under one of the many problems muskuloskeletal (eg regangan lumbosakral acute, ligamen lumbosakral instability and muscle weakness, osteoartritis spine, spinal stenosis, a problem intervertebralis discus, leg length inequality). Other causes include Obesity, kidney disturbances, problems pelvis, retroperitoneal tumor, aneurisma abdominal and psychosomatic problems. Most back pain due to interference by muskuloskeletal akan diperberat activity, whereas pain due to other circumstances is not affected by the activity (2,4).

C. Patofisiology
Specific structure in the nervous system involved in the stimulus became painful sensation. System involved in pain transmission and perception is called the system nosiseptif. Sensitivity of the system components nosiseptif can be influenced by a number of factors and vary among individuals. Not all those who terpajan to the same stimulus intensity experienced the same pain. Sensation is very painful for someone may not feel close to others (1,3).
Reseptor pain (nosiseptor) is the free end of the nerve in the skin berespons only on a strong stimulus, which is potentially harmful, which are the stimuli can be chemical, mechanical, thermal. Reseptor pain is a multi-way jaras complex. Nerve fiber is very close to the fork originally on the skin and sends branches to the local blood vessel. Mast cells, folikel hair and sweat gland. Stimuli fiber histamin this result in release from mast cells and cause vasodilatasi. Fiber kutaneus located more towards the central branch of the further and related to the chain simpatis paravertebra nervous system and internal organs with a larger. A number of substances that can increase the transmission or perception of pain include histamin, bradikinin, asetilkolin and substance P. Prostaglandin where substances that can increase the effect of that cause pain bradikinin. Other substances in the body that functions as a inhibitor of pain transmission is endorfin and enkefalin found in the concentration of a strong central nervous system (1,3).
Kornu dorsalis of the medulla spinalis is a sensori process, which can be painful to be consciously, in neuron system assenden must be enabled. Activation occurs as a result of input from the painful reseptor located in the skin and internal organs. The process occurs because of a painful interaction between pain stimulus and pain sensation (1,3).
In the painful sensation Patofisiologi back down in this case kolumna vertebralis can be considered as an elastic rod is composed of many units on the vertebrae and the discus intervertebrae tied to one another by complex faset joints, muscles and ligamen various paravertebralis. Back the unique construction allows flexibility while on the other can still memberikanperlindungan the sum of the maximum-sum spine. Spinal curvature akan absorb shock when the vertical run or jump. Torso help stabilize the spine. Abdominal musculature and have very important Toraks activities lift the burden. When not in use will never weaken the structure of this support. Obesity, the postures, the problem of structure and support peregangan excessive spine can result in back pain (2,4).
Discus intervertebralis akan changing nature of the increasing old age. In the young people, especially structured discus fibrokartilago up with gelatinus matrix. In the elderly will become a solid and fibrokartilago ramble. Discus intervertebra degeneration is a common cause of back pain. Discus lumbal down, L4-L5 and L5-S6, suffer the most stress and weight change terberat degeneration. Projection discus or damaged joints can lead to emphasis on the nerve roots exit kanalis spinalis, causing pain that spreads along the nerve (2,4).

D. Clinical manifestations
Patients usually engeluh painful punngung acute and chronic back pain and weakness. During the initial interview locations painful knowledge, and are penjalarannya along the nerve fiber (sciatica), I also evaluated the way patients, spinal mobility, reflex, leg length, strength and motorist perception sensoris with the degree of inconvenience dialaminya. Rise straight leg in a show cause painful irritation nerve fiber.
Physical examination can find the spasme muscle paravertebralis (increased bone postural muscle tonus back over), along with loss of flexion lordotik lumbal normal and that there may be deformitas spine. When the patient is examined in telungkup, akan paraspinal muscle relaxation and deformitas caused by spasme akan disappeared.
Sometimes basic organic back pain can not be found. Worries and stress can raise spasme and muscle aches. Pain down the back can be anifestasi depression or mental conflict or reaction to a stressor environment and life. When we examine patients with painful punngung down, nurses need to review the family relationship, a variable environment and work situation (2,4).

E. Diagnostic Evaluation
Diagnostic procedures should be performed on patients who mendertita sore back down. X-rays showed the vertebra may fraktur, dislocation, infection, scoliosis or osteoartritis. Computed Tomografi (CT) is useful to know that underlie the disease, such as the network software lesi hidden around the kolumna vertebralis and discus intervertebralis problem. USG can help diagnose kanalis constringency spinalis. MRI allows visualization nature and location of spinal pathology (2).


F. Implementations
Most back pain can be lost and will recover in 6 weeks with tirah lie, stress reduction and relaxation. Patients must stay at a bed with mattress and solid membal not for 2 to 3 days. Position the patient so that made FLEKSI lumbal greater that can reduce the pressure on the nerve fiber lumbal. The head of the bed elevated 30 degrees and slightly bend patients lie lututnya or italics with lutu and ditekuk pelvis and leg and a pillow placed under the head. Prone position because it will aggravate avoided lordosis. Sometimes patients need to be treated for the handling of "active conservative" and physiotherapy. Traksi pelvic intermiten with 7 to 13 kg load traksi. Traksi allows the addition FLEKSI lumbal and muscle relaxation is.
Physiotherapy should be given to reduce pain and muscle spasme. Therapy may include cooling (with ice missal), heating infra red rays, compress and humid summer, swimming and turbulent traksi. Circulation disturbances, disruption and trauma palpability is contra indications hot compress. Turbulent dikontraindikasikan therapy pool for patients with problems due to the inability kardiovaskuler tolerate vasodilatasi perifer massif that arise. Ultra will cause waves that heat can increase the inconvenience caused swelling in the acute stages.
Drugs may be needed to deal with acute pain. Analgetik drug used to cut off the circle aches, muscle relaksan tranquilizer and used to make relaks the patient and the spasme muscle, which can reduce the pain. Antiinflamasi drugs, such as aspirin and antiinflamasi nonsteroid drugs (NSAID), is useful for reducing pain. Kortikosteroid short-term response inflamasi can reduce and prevent the emergence neurofibrosis that occur due to interference iskemia (2,4).

G. Anamnesis
Patients pungung pain led to the inconvenience (missal location, weight, duration, nature, spread and leg weakness associated). Explanation of how the pain relief with a certain action or activity where the muscles are weak over-used and how patients cope. Information on work and recreational activities can help identify areas for health education.
During this interview, the nurse can make observations of the patient postures, the position deviation and how the road. On physical examination, examined spinal curvature, Krista iliakan and kesimetrisan shoulder. Paraspinal muscle dipalpasi and note the existence of pain and spasme press. Patients reviewed the Obesity can be caused due to back pain below (2).

H. Nursing Diagnosis (2)
1.Nyeri b.d problem muskuloskeletal
2.Kerusakan bd painful physical mobility, spasme muscle, and less kelenturan
Bd 3.Kurang knowledge engineering mechanics to protect the body back
4.Perubahan performance of the role of mobility and interference bd painful chronicles
5.Gangguan nutrition: more than the needs of the body b. d Obesity

I. Intervention and Implementation (2)
Pain reduction
To reduce the pain nurse can recommend tirah recline position and conversion, which is determined to improve FLEKSI lumbal. Taught patients to control and adjust the pain through the respiratory diaphragm and relaxation can help reduce muscle tension that contribute to the pain back down. Distract attention from the patient's pain with other activities missal to read books, watch TV and the imagination (imagine excitement with the focus on it).
Massage with a soft network software is very useful to reduce spasme muscles, improve blood circulation and reduce pain and reduce pembendungan. When nurses are given the medicine must examine the response of each patient on medication.

recovery physical mobility
Physical mobility of monitored through continuous. How nurses assess patients' movement and standing. So back pain is reduced, self-care activities may be done with a minimal regangan on the structure of the injury. Changes in position should be done slowly and dibatu if necessary. Movement and play should be avoided sway. Patients are encouraged to take turns aktifiats lie, sit and walk in a long time. Nurses need to encourage patients to comply with the appropriate training program is set, a practice that is not effective.

increase the appropriate body mechanics
Patients should be taught how to sit, stand, stretch and lift items correctly.

health educations
Patients should be taught how to sit, stand, stretch and lift items correctly

role performance recover
Responsibilities related to the role may have changed since the occurrence of pain down the back. Once the sore healed, the patient can return to the responsibility of the role again. However, when these activities affect the occurrence of painful pungung down again, may be difficult to return to the responsibility of the bear without the risk of a painful chronicle pungggung down with a disability and depression caused.

changes nutrition and a decrease in body weight
BB through a decrease in how meals adjustment kekambuhan can prevent back pain, with nutrition through a rational plan that includes changes in eating kebaisaaan to maintain the desired BB.

J. Evaluation (2)
quieting pain
Resting comfortably
Comfortable with the position change
Avoiding drug dependency

return of physical mobility
Return to activity gradually
Avoiding positions that cause the muscles that cause inconvenience
Plan rest recline all day

showing body mechanics of the spine
Repairs postures
Changing the position itself to minimize back stress
Demonstrate the use of good body mechanics
Participating in the exercise

back to the responsibilities associated with the role
Using the techniques have problems to adapt to the stressful situation
Shows the reduced dependence to another person for self-care
Back to work when the back pain was healed
Back to the lifestyle of full and productive

desired body weight
Identify the need for a decrease in BB
Participate in the development plan reduction BB
Reduction program with a loyal BB

References:
1.Brunner & Suddarth, Change Language Monica Esther, SKP; Book Ajar medical Surgical Nursing, 8 Edition, Volume 1, EGC, Jakarta, 2002
2.Brunner & Suddarth, Change Language Monica Esther, SKP; Book Ajar medical Surgical Nursing, 8 Edition, Volume 3, EGC, Jakarta, 2002
3.Ruth F. Craven, EdD, RN, Fundamentals Of Nursing, Edition II, Lippincot, Philadelphia, 2000
4.Wim de Jong, Buku Ajar Surgical Sciences, Revised Edition, Printed I, EGC, Jakarta, 1997
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